Research Area

The research in my lab is focused on understanding the lipid metabolic pathways that enable Mycobacterium tuberculosis, the causative agent of tuberculosis, to enter a state of dormancy and become tolerant to antibiotics resulting in latent infection. Our ongoing studies aim to functionally characterize the genes involved in the accumulation of energy reserves as lipids in the bacterial pathogen during its dormancy. We seek to understand the biochemical functions of mycobacterial proteins associated with lipid droplet homeostasis and storage lipid metabolism.


Opportunities for graduate and undergraduate students to participate in the ongoing research projects in my laboratory are available. Contact me by email ( learn more.